Ca2+/Calmodulin-dependent Protein Kinase II Inhibitors Disrupt AKAP79-dependent PKC Signaling to GluA1 AMPA Receptors*
Journal of Biological Chemistry（2010）
Key words
Calmodulin,Calcium/Calmodulin-dependent Protein Kinase (CaMK),Drug Action,Glutamate Receptors, Ionotropic (AMPA, NMDA),Protein Kinase C (PKC),Protein Phosphorylation,AKAP,LTP,Synaptic Plasticity
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