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Loss of Tmem106b is Unable to Ameliorate Frontotemporal Dementia-Like Phenotypes in an AAV Mouse Model of C9ORF72-repeat Induced Toxicity

Alexandra M. Nicholson,Xiaolai Zhou,Ralph B. Perkerson,Tammee M. Parsons,Jeannie Chew,Mieu Brooks,Mariely DeJesus-Hernandez,NiCole A. Finch,Billie J. Matchett,Aishe Kurti,Karen R. Jansen-West,Emilie Perkerson,Lillian Daughrity,Monica Castanedes-Casey,Linda Rousseau,Virginia Phillips,Fenghua Hu,Tania F. Gendron,Melissa E. Murray,Dennis W. Dickson,John D. Fryer,Leonard Petrucelli,Rosa Rademakers

Acta Neuropathologica Communications（2018）

Cited 18|Views76
Key words
C9ORF72 Repeat,Transmembrane Protein 106B (TMEM106B),Chromosome 9 Open Reading Frame 72 (C9ORF72),Dipeptide Repeat Proteins,C9ORF72 Expansion
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