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HNF4A and GATA6 Loss Reveals Therapeutically Actionable Subtypes in Pancreatic Cancer

Holly Brunton,Giuseppina Caligiuri,Richard Cunningham,Rosie Upstill-Goddard,Ulla-Maja Bailey,Ian M. Garner,Craig Nourse,Stephan Dreyer,Marc Jones,Kim Moran-Jones,Derek W. Wright,Viola Paulus-Hock,Colin Nixon,Gemma Thomson,Nigel B. Jamieson, Grant A. McGregor,Lisa Evers,Colin J. McKay,Aditi Gulati,Rachel Brough,Ilirjana Bajrami,Stephen J. Pettitt,Michele L. Dziubinski,Simon T. Barry,Robert Gruetzmann, Robert Brown,Edward Curry,Marina Pajic,Elizabeth A. Musgrove,Gloria M. Petersen,Emma Shanks,Alan Ashworth,Howard C. Crawford,Diane M. Simeone,Fieke E. M. Froeling,Christopher J. Lord,Debabrata Mukhopadhyay,Christian Pilarsky,Sean E. Grimmond,Jennifer P. Morton,Owen J. Sansom,David K. Chang,Peter J. Bailey,Andrew Biankin

SSRN Electronic Journal（2019）

Cited 78|Views74
Key words
PDAC subtypes,HNF4A,GATA6,metabolic targeting,GSK3B,intronic and distal promoters,chromatin landscapes,therapeutic tolerance
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