Supplementary Data from Insight into the Etiology of Undifferentiated Soft Tissue Sarcomas from a Novel Mouse Model

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摘要
Figure S1. PCG2 tumors showed persistent Hh pathway activity, while SmoM2 disrupts brain morphogenesis. Figure S2. PCG2 gene expression and ultrastructural features. Figure S3. Characterization of PCG2M226 cells. Figure S4. Phenotypic divergence between PCG2; Gli1nlacZ and PCG2 tumors. Figure S5. Fate-mapping of Pcp2-cre lineage cells.
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