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Cell-autonomous IL6ST Activation Suppresses Prostate Cancer Development Via STAT3/ARF/p53-driven Senescence and Confers an Immune-Active Tumor Microenvironment

Christina Sternberg,Martin Raigel,Tanja Limberger,Karolína Trachtová,Michaela Schlederer, Desiree Lindner,Petra Kodajova,Jiaye Yang,Roman Ziegler,Jessica Kalla,Stefan Stoiber,Saptaswa Dey,Daniela Zwolanek,Heidi A. Neubauer,Monika Oberhuber,Torben Redmer,Václav Hejret,Boris Tichy,Martina Tomberger,Nora S. Harbusch,Jan Pencik,Simone Tangermann,Vojtech Bystry,Jenny L. Persson,Gerda Egger,Sarka Pospisilova,Robert Eferl,Peter Wolf,Felix Sternberg,Sandra Högler,Sabine Lagger,Stefan Rose-John,Lukas Kenner

Molecular Cancer（2024）

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Key words
Prostate cancer,IL6ST/STAT3 signaling,L-gp130,Senescence,Senescence-associated secretory phenotype,Tumor microenvironment,Immune cell infiltration,Cytotoxic T-cells
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